Indications

• Cellular growth & differentiation
• Antioxidant
• Immunity

Ingredients

Serving Size: 1 Capsule... %DV
Selenium (as Selenomethionine)... 55 mcg... 79%

Description

What Is It?
SeMet is selenomethionine, a natural form of selenium that is estimated to make up about half of the selenium obtained from dietary sources. It is the main form in which the body stores selenium, and is easily used by the body and has been shown to have a bioavailability at least 1.5-2 times higher than inorganic forms of selenium.

What Does It Do?
Primary Uses: Research has linked selenium-rich soil with lower cancer rates, and selenium is thought to help control the formation of abnormal cells. But not all forms of selenium are equal. SeMet is less toxic and more potent than other forms of selenium. Selenium's protective potential is linked to its formation of methylselenol. Selenomethionine is directly and easily converted into this important metabolite.
Secondary Uses: Selenium is necessary for immune function, supporting the formation of the enzyme glutathione peroxidase, a powerful antioxidant, and boosting immune cell activity.

Who Should Take It?
Selenium is an important trace mineral, especially where soil levels of selenium are depleted, and can provide support for those concerned with healthy cellular growth and good immune function.

Quantity

90 Vegi-Caps

Dose

Take 1 capsule daily with a meal, or as directed by a qualified health care practitioner.

Potential side effects/Safety

7191

References

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Ip C, Hayes C, Budnick RM, Ganther HE. “
Chemical form of selenium, critical metabolites, and cancer prevention.”

Cancer Res 1991 Jan 15; 51(2): 595-600.

Ip C. “
Lessons from basic research in selenium and cancer prevention.”

J Nutr. 1998 Nov; 128(11): 1845-54.

Limburg P.J. et al. “Randomized, Placebo-Controlled, Esophageal Squamous Cell Cancer Chemoprevention Trial of Selenomethionine and Celecoxib.” Gastroenterology. 2005. 129(3): 863-873.

Schrauzer G.N. “Selenomethionine: A Review of Its Nutritional Significance, Metabolism and Toxicity. Journal of Nutrition”. 2000. 130: 1653-1656.

Spallholz J.E. et al. Methioninase and selenomethionine but not Se-methylselenocysteine generate methylselenol and superoxide in an in vitro chemiluminescent assay: implications for the nutritional carcinostatic activity of selenoamino acids. Biochemical Pharmacology. 2004. 67(3): 547-554.

Whanger PD. “
Selenocompounds in plants and animals and their biological significance.”

J Am Coll Nutr. 2002 Jun; 21(3): 223-32.